Most noteworthy for me is the fact that more Americans think that porn (52%) and homosexuality (39%) are wrong than spanking children (23%) and being ultra wealthy (18%).
🤡 country
https://www.pewresearch.org/religion/2026/03/19/what-do-americans-consider-immoral/
I saw it first hand. Friend lost his wife and house to weed. Two other people in our circles had weed psychosis. Thankfully early intervention helped them recover. But we also know one person that went into it never recovered. And became a danger to those around him. Now he’s in jail.
It happens with alcohol too, but not the everlasting schizo issues
Eh, I’d bet alcohol is more on the everlasting schizophrenia risk side. But weed really does fuck up your thinking.
Like every occasional sixth month or year in a conducive environment, yes, it may enhance thinking outside the box for an insight or few. Daily would be like a milder version of dropping hallucinogens.
Studies have shown a direct link to weed induced schizo disorders and those with genetic markers susceptible for it. Getting a DNA test is not on most peoples priority list.
I’m not into scare mongering, either. The incidence of schizophrenia is very low.
https://ourworldindata.org/grapher/schizophrenia-prevalence
I think the science paper I read was 7x higher rate on weed, ifyour had a certain gene. I will have to find it
That’s still not statistically significant, and correlation does not imply causation. It serves as a caution, and I’m inclined to agree it’s not wise to risk it, but let’s be honest with ourselves and others.
Well correlation studiear say 37% risk, but I recognize that correlation is not causation, however they have found the mechanism that causes the increased psychosis with genes.
https://www.mdpi.com/2076-3417/12/20/10464
AKT1 gene polymorphism rs2494732 as the main genetic factor
C allele homozygosity significantly increases psychosis risk in cannabis users.
Mechanism: Cannabis consumption slows AKT1 functionality, releasing greater dopamine in the striatum through indirect mechanisms.
COMT and DAT1 gene interactions compound the risk
CB-1 receptor activation leading to increased dopamine release and GABA inhibition